Bisphenol A accelerates meiotic progression in embryonic chickens via the estrogen receptor β signaling pathway-Reference-Cited by-同舟云学术

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Bisphenol A accelerates meiotic progression in embryonic chickens via the estrogen receptor β signaling pathway

Published:2018-04 Issue: Volume:259 Page:66-75
ISSN:0016-6480
Container-title:General and Comparative Endocrinology
language:en
Short-container-title:General and Comparative Endocrinology

Author:

Yu Minli,Xu Yali,Li Ming,Li Dongfeng,Lu Yinglin,Yu Debing,Du Wenxing

Funder

National Natural Science Foundation of China

Fundamental Research Funds for the Central Universities

Technology Innovation Foundation by Nanjing Agriculture University

National Science and Technology Support Program

Publisher

Elsevier BV

Subject

Endocrinology,Animal Science and Zoology

Reference33 articles.

1. Low doses of bisphenol A promote human seminoma cell proliferation by activating PKA and PKG via a membrane G-protein-coupled estrogen receptor;Bouskine;Environ. Health Perspect.,2009

2. Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response;Bromer;FASEB J.,2010

3. Bisphenol A exposure modifies methylation of imprinted genes in mouse oocytes via the estrogen receptor signaling pathway;Chao;Histochem. Cell Biol.,2012

4. Bisphenol a exposure: human risk and health policy;Erler;J. Pediatr. Nurs.,2010

5. Bisphenol A-induced ovotoxicity involves DNA damage induction to which the ovary mounts a protective response indicated by increased expression of proteins involved in dna repair and xenobiotic biotransformation;Ganesan;Toxicol. Sci.,2016

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