The adenosine inhibition of glutamate exocytosis in synaptosomes is removed by the collapse of the vesicle–cytosol ΔpH plus the opening of farnesol-sensitive Ca2+ channels-Reference-Cited by-同舟云学术

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The adenosine inhibition of glutamate exocytosis in synaptosomes is removed by the collapse of the vesicle–cytosol ΔpH plus the opening of farnesol-sensitive Ca2+ channels

Published:2003-04 Issue:4 Volume:33 Page:273-282
ISSN:0143-4160
Container-title:Cell Calcium
language:en
Short-container-title:Cell Calcium

Author:

Zoccarato F.,Cavallini L.,Alexandre A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference38 articles.

1. The glutamatergic nerve terminal;Nicholls;Eur. J. Biochem.,1993

2. Presynaptic receptors and the control of glutamate exocytosis;Sanchez-Prieto;Trends Neurosci.,1996

3. Aminoacid neurotransmission: spotlight on synaptic vesicles;Maycox;Trends Neurosci.,1990

4. The role of v-ATPase in neuronal and endocrine systems;Moriyama;J. Exp. Biol.,1992

5. Organellar proton-ATPases;Nelson;Curr. Opin. Cell Biol.,1992

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Calcitox-aging counterbalanced by endogenous farnesol-like sesquiterpenoids: An undervalued evolutionarily ancient key signaling pathway;Communicative & Integrative Biology;2017-07-04

2. Synaptic vesicle cycling is not impaired in a glutamatergic and a cholinergic synapse that exhibit deficits in acidification and filling;Brazilian Journal of Pharmaceutical Sciences;2012-03

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