JAB1/CSN5 inhibits the activity of Luman/CREB3 by promoting its degradation
Author:
Publisher
Elsevier BV
Subject
Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Reference58 articles.
1. Luman, a new member of the CREB/ATF family, binds to herpes simplex virus VP16-associated host cellular factor;Lu;Mol. Cell. Biol.,1997
2. Viral mimicry: common mode of association with HCF by VP16 and the cellular protein LZIP;Freiman;Genes Dev.,1997
3. Purification of the cellular C1 factor required for the stable recognition of the Oct-1 homeodomain by the herpes simplex virus alpha-trans-induction factor (VP16);Kristie;J. Biol. Chem.,1993
4. The VP16 accessory protein HCF is a family of polypeptides processed from a large precursor protein;Wilson;Cell,1993
5. The herpesvirus transactivator VP16 mimics a human basic domain leucine zipper protein, Luman, in its interaction with HCF;Lu;J. Virol.,1998
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1. The stability of CREB3/Luman is regulated by protein kinase CK2 phosphorylation;Biochemical and Biophysical Research Communications;2020-03
2. Axotomy Induces Phasic Alterations in Luman/CREB3 Expression and Nuclear Localization in Injured and Contralateral Uninjured Sensory Neurons: Correlation With Intrinsic Axon Growth Capacity;Journal of Neuropathology & Experimental Neurology;2019-03-12
3. A CREB3–ARF4 signalling pathway mediates the response to Golgi stress and susceptibility to pathogens;Nature Cell Biology;2013-11-03
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