Effect of simvastatin on monocyte adhesion molecule expression in patients with hypercholesterolemia-Reference-Cited by-同舟云学术

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Effect of simvastatin on monocyte adhesion molecule expression in patients with hypercholesterolemia

Published:2001-08 Issue:2 Volume:157 Page:505-512
ISSN:0021-9150
Container-title:Atherosclerosis
language:en
Short-container-title:Atherosclerosis

Author:

Serrano Jr. Carlos V,Yoshida Vanda M,Venturinelli Margareth L,D'Amico Elbio,Monteiro Hugo P,Ramires José Antônio F,da Luz Protásio L

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference34 articles.

1. Antiatherothrombotic properties of statins: implications for cardiovascular event reduction;Rosenson;JAMA,1998

2. Oxidized low density lipoprotein induces differentiation and adhesion of human monocytes and the monocytic cell line U937;Frostegård;Proc Natl Acad Sci USA,1990

3. Effect of cholesterol lowering treatment on positive exercise tests in patients with hypercholesterolaemia and normal coronary angiograms;Mansur;Heart,1999

4. Traffic signals on endothelium for leukocytes in health, inflammation, and atherosclerosis;Springer,1996

5. Adhesion molecules – part II: blood vessels and blood cells;Frenette;N Eng J Med,1996

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1. Chronic statin treatment is a predictor of pre-interventional infarct-related artery patency in patients with ST elevation myocardial infarction treated with percutaneous coronary intervention;Kardiologia Polska;2018-03-16

2. Identification of cardiovascular risk factors associated with bone marrow cell subsets in patients with STEMI: a biorepository evaluation from the CCTRN TIME and LateTIME clinical trials;Basic Research in Cardiology;2016-11-23

3. The Role of Different Monocyte Subsets in the Pathogenesis of Atherosclerosis and Acute Coronary Syndromes;Scandinavian Journal of Immunology;2015-08-18

4. Trans-vessel gradient of myeloperoxidase in coronary artery disease;European Journal of Clinical Investigation;2013-07-22

5. Short-term therapy with simvastatin reduces inflammatory mediators and heart inflammation during the acute phase of experimental Chagas disease;Memórias do Instituto Oswaldo Cruz;2012-06

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