Motor disturbances in mice with deficiency of the sodium channel gene Scn8a show features of human dystonia-Reference-Cited by-同舟云学术

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Motor disturbances in mice with deficiency of the sodium channel gene Scn8a show features of human dystonia

Published:2003-12 Issue:2 Volume:184 Page:830-838
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Hamann Melanie,Meisler Miriam H,Richter Angelika

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference48 articles.

1. Dopamine transmission in DYT1 dystonia: a biochemical and autoradiographical study;Augood;Neurology,2002

2. Clinical-genetic spectrum of primary dystonia;Bressman;Adv. Neurol.,1998

3. Sodium channel Nav1.6 is localized at nodes of Ranvier, dendrites, and synapses;Caldwell;Proc. Natl. Acad. Sci. U. S. A.,2000

4. What's wrong with my mouse?;Crawley,2000

5. Paroxysmal dyskinesias: clinical features and classification;Demirkiran;Ann. Neurol.,1995

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