Mitochondrial alterations triggered by repeated exposure to fine (PM2.5-0.18) and quasi-ultrafine (PM0.18) fractions of ambient particulate matter-Reference-Cited by-同舟云学术

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Mitochondrial alterations triggered by repeated exposure to fine (PM2.5-0.18) and quasi-ultrafine (PM0.18) fractions of ambient particulate matter

Published:2020-09 Issue: Volume:142 Page:105830
ISSN:0160-4120
Container-title:Environment International
language:en
Short-container-title:Environment International

Author:

Sotty J.,Kluza J.,De Sousa C.,Tardivel M.,Anthérieu S.,Alleman L.-Y.,Canivet L.,Perdrix E.,Loyens A.,Marchetti P.,Lo Guidice J.-M.,Garçon G.

Funder

Région Hauts-de-France

Publisher

Elsevier BV

Subject

General Environmental Science

Reference98 articles.

1. Air pollution Particulate Matter (PM2,5)-induced gene expression of volatile organic compound and/or polycyclic aromatic hydrocarbon-metabolizing enzymes in an in vitro coculture lung model;Abbas;Toxicol. In Vitro,2009

2. Occurrence of molecular abnormalities of cell cycle in L132 cells after in vitro short-term exposure to air pollution PM2,5;Abbas;Chem. Biol. Interact.,2010

3. Polycyclic aromatic hydrocarbons within airborne particulate matter (PM2,5) produced DNA bulky stable adducts in a human lung cell coculture model;Abbas;J. Appl. Toxicol.,2013

4. In vitro short-term exposure to air pollution PM2,5–0,3 induced cell cycle alterations and genetic instability in a human lung cell coculture model;Abbas;Env. Res.,2016

5. In vitro evaluation of organic extractable matter from ambient PM2.5 using human bronchial epithelial BEAS-2B cells: Cytotoxicity, oxidative stress, pro-inflammatory response, genotoxicity, and cell cycle deregulation;Abbas;Environ. Res.,2019

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