Deletion of liver-specific STAT5 gene alters the expression of bile acid metabolism genes and reduces liver damage in lithogenic diet-fed mice

Author:

Baik Myunggi,Kim Jangseon,Piao Min Yu,Kang Hyeok Joong,Park Seung Ju,Na Sang Weon,Ahn Sung-HoonORCID,Lee Jae-Hyuk

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Nutrition and Dietetics,Molecular Biology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference29 articles.

1. Growth hormone activation of Stat 1, Stat 3, and Stat 5 in rat liver. Differential kinetics of hormone desensitization and growth hormone stimulation of both tyrosine phosphorylation and serine/threonine phosphorylation;Ram;J Biol Chem,1996

2. The role of the growth hormone (GH) receptor and JAK1 and JAK2 kinases in the activation of Stats 1, 3, and 5 by GH;Smit;Mol Endocrinol,1996

3. GH-dependent STAT5 signaling plays an important role in hepatic lipid metabolism;Barclay;Endocrinology,2011

4. Liver-specific deletion of the signal transducer and activator of transcription 5 gene aggravates fatty liver in response to a high-fat diet in mice;Baik;J Nutr Biochem,2016

5. Growth hormone treatment does not alter biliary lipid metabolism in healthy adult man;Olivecrona;J Clin Endocrinol Metab,1995

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