Dietary polyunsaturated fatty acids suppress acute hepatitis, alter gene expression and prolong survival of female Long-Evans Cinnamon rats, a model of Wilson disease

Author:

Du Chunyan,Fujii Yoichi,Ito Masafumi,Harada Manabu,Moriyama Emiko,Shimada Ryo,Ikemoto Atsushi,Okuyama* Harumi

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Nutrition and Dietetics,Molecular Biology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference38 articles.

1. High susceptibility to hepatocellular carcinoma development in LEC rats with hereditary hepatitis;Masuda;Jpn J Cancer Res,1988

2. Phenotype of preneoplastic and neoplastic liver lesions during spontaneous liver carcinogenesis of LEC rats;Sawaki;Carcinogenesis,1990

3. Fetal copper uptake and a homolog (Atp7b) of the Wilson's disease gene in rats;Muramatsu;Res Commun Mol Pathol Pharmacol,1998

4. Spontaneous hepatic copper accumulation in Long-Evans Cinnamon rats with hereditary hepatitis. A model of Wilson's disease;Li;J Clin Invest,1991

5. Decrease in biliary excretion of copper in Long-Evans Cinnamon (LEC) rats causing spontaneous hepatitis due to a gross accumulation of hepatic copper;Sugawara;Res Commun Chem Pathol Pharmacol,1993

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