Correction of erythrocyte abnormalities in idiopathic calcium-oxalate nephrolithiasis and reduction of urinary oxalate by oral glycosaminoglycans
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference20 articles.
1. Raised transmembrane oxalate flux in red blood cells in idiopathic calcium oxalate nephrolithiasis;Baggio;Lancet,1984
2. An inheritable anomaly of red-cell oxalate transport in "primary" calcium nephrolithiasis correctable with diuretics;Baggio;N Engl J Med,1986
3. Relation between band 3 red blood cell protein and transmembrane oxalate flux in stone formers;Baggio;Lancet,1984
4. Altered red blood cell membrane protein phosphorylation in idiopathic calcium oxalate nephrolithiasis;Baggio;IRCS Med Sci,1986
5. Glycosaminoglycan content, oxalate self-exchange and protein phosphorylation in erythrocytes of patients with "idiopathic" calcium oxalate nephrolithiasis;Baggio;Clin Sci,1990
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1. Modulators of urinary stone formation;Frontiers in Bioscience;2004
2. A complementary approach to urolithiasis prevention;World Journal of Urology;2002-11
3. Urinary Kidney Stone Inhibitors. What Is the New?;Urologia Internationalis;1998
4. Pathogenesis of Idiopathic Calcium Nephrolithiasis: Update 1997;Critical Reviews in Clinical Laboratory Sciences;1998-01
5. Glycosaminoglycans and semisynthetic sulfated polysaccharides: An overview of their potential application in treatment of patients with urolithiasis;Urology;1997-08
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