Circ_0005699 participates in ox-LDL-induced human umbilical vein endothelial cell injury via targeting the miR-636/TLR4/NF-κB pathway-Reference-Cited by-同舟云学术

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Circ_0005699 participates in ox-LDL-induced human umbilical vein endothelial cell injury via targeting the miR-636/TLR4/NF-κB pathway

Published:2022-08 Issue: Volume:186 Page:108579
ISSN:1369-703X
Container-title:Biochemical Engineering Journal
language:en
Short-container-title:Biochemical Engineering Journal

Author:

Tao Guilu,Zhong Cundi,Zhang Lei,Zhong Xiaoguang,Li Xiaolu,Wang Zhaoyun,Xu Yunpeng,Zhao Yiqun,Mao Ping

Publisher

Elsevier BV

Subject

Biomedical Engineering,Environmental Engineering,Bioengineering,Biotechnology

Reference46 articles.

1. Oxidized low density lipoproteins: the bridge between atherosclerosis and autoimmunity. Possible implications in accelerated atherosclerosis and for immune intervention in autoimmune rheumatic disorders;Suciu;Autoimmun. Rev.,2018

2. An activator of mTOR inhibits oxLDL-induced autophagy and apoptosis in vascular endothelial cells and restricts atherosclerosis in apolipoprotein E(-)/(-) mice;Peng;Sci. Rep.,2014

3. Endothelial cell dysfunction as a novel therapeutic target in atherosclerosis;Jensen;Expert Rev. Cardiovasc Ther.,2016

4. Ras induces vascular smooth muscle cell senescence and inflammation in human atherosclerosis;Minamino;Circulation,2003

5. Salidroside slows the progression of EA.hy926 cell senescence by regulating the cell cycle in an atherosclerosis model;Sun;Mol. Med. Rep.,2018

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1. NLRP3 inflammasome pathway in atherosclerosis: Focusing on the therapeutic potential of non-coding RNAs;Pathology - Research and Practice;2023-06

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