Effects of valproate, vigabatrin and aminooxyacetic acid on release of endogenous and exogenous GABA from cultured neurons

Author:

Gram L.,Larsson O.M.,Johnsen A.H.,Schousboe A.

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology

Reference57 articles.

1. The action of γ-vinyl GABA and γ-acetylenic GABA on the resting and stimulated release of GABA in vivo;Abdul-Ghani;Brain Res.,1980

2. Effect of γ-acetylenic GABA and γ-vinyl GABA on synaptosomal release and uptake of GABA;Abdul-Ghani;Biochem. Pharmacol.,1981

3. On the existence of two GABA pools associated with newly synthesized GABA and with newly taken up GABA in nerve terminals;Abe;Neurochem. Res.,1983

4. Sodium valproate enhancement of γ-aminobutyric acid (GABA) inhibition: electrophysiological evidence for anticonvulsant activity;Baldino;J. Pharmacol. Exp. Ther.,1981

5. Mechanism of anticonvulsant action of valproate;Chapman;Prog. Neurobiol.,1982

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