Dominant role of mitochondria in protection against a delayed neuronal Ca2+ overload induced by endogenous excitatory amino acids following a glutamate pulse-Reference-Cited by-同舟云学术

Dominant role of mitochondria in protection against a delayed neuronal Ca2+ overload induced by endogenous excitatory amino acids following a glutamate pulse

Published:1996-09-09 Issue:1 Volume:393 Page:135-138
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Khodorov B.,Pinelis V.,Storozhevykh T.,Vergun O.,Vinskaya N.

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(96)00873-3/fullpdf

Reference13 articles.

1. Mitochondria and Na+/Ca2+ exchange buffer glutamate-induced calcium loads in cultured cortical neurons

2. A Reevaluation of the Role of Mitochondria in Neuronal Ca2+ Homeostasis

3. Inhibition of Na+/Ca2+ exchange enhances delayed neuronal death elicited by glutamate in cerebellar granule cell cultures

4. Dramatic effects of external alkalinity on neuronal calcium recovery following a short-duration glutamate challenge: the role of the plasma membrane Ca2+/H+pump

5. Glutamate impairs neuronal calcium extrusion while reducing sodium gradient

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