Identification of in vivo phosphorylation sites of SET, a nuclear phosphoprotein encoded by the translocation breakpoint in acute undifferentiated leukemia
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(94)80144-4/fullpdf
Reference28 articles.
1. The translocation (6;9), associated with a specific subtype of acute myeloid leukemia, results in the fusion of two genes, dek and can, and the expression of a chimeric, leukemia-specific dek-can mRNA.
2. Can, a putative oncogene associated with myeloid leukemogenesis, may be activated by fusion of its 3' half to different genes: characterization of the set gene.
3. Identification and characterization of SET, a nuclear phosphoprotein encoded by the translocation break point in acute undifferentiated leukemia.
4. Identification and molecular cloning of yeast homolog of nucleosome assembly protein I which facilitates nucleosome assembly in vitro.
5. Structure of a gene coding for human HMG2 protein.
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1. Protocadherin-7 Regulates Osteoclast Differentiation through Intracellular SET-Binding Domain-Mediated RhoA and Rac1 Activation;International Journal of Molecular Sciences;2021-12-04
2. SET Protein in Cancer: A Potential Therapeutic Target;Mini-Reviews in Medicinal Chemistry;2021-11-23
3. Targeting of SET/I2PP2A oncoprotein inhibits Gli1 transcription revealing a new modulator of Hedgehog signaling;Scientific Reports;2021-07-06
4. The Key Role of the Phosphatase PP2A in the Development of Acute Myeloid Leukemia;Acute Leukemias;2021-03-24
5. PP2A-B55 Holoenzyme Regulation and Cancer;Biomolecules;2020-11-22
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