Analysis of PI (phosphatidylinositol)-anchoring antigens in a patient of paroxysmal nocturnal hemoglobinuria (PNH) reveals deficiency of 1F5 antigen (CD59), a new complement-regulatory factor
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(90)80656-4/fullpdf
Reference28 articles.
1. Affected erythrocytes of patients with paroxysmal nocturnal hemoglobinuria are deficient in the complement regulatory protein, decay accelerating factor.
2. Deficiency of an erythrocyte membrane protein with complement regulatory activity in paroxysmal nocturnal hemoglobinuria.
3. Two populations of erythroid cell progenitors in paroxysmal nocturnal hemoglobinuria
4. Deficiency of the Complement Regulatory Protein, Decay-Accelerating Factor, on Membranes of Granulocytes, Monocytes, and Platelets in Paroxysmal Nocturnal Hemoglobinuria
5. Distribution of decay-accelerating factor in the peripheral blood of normal individuals and patients with paroxysmal nocturnal hemoglobinuria.
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1. Inherited Complement Deficiencies;Emery and Rimoin's Principles and Practice of Medical Genetics and Genomics;2023
2. Increased Expression of Complement Regulators CD55 and CD59 on Peripheral Blood Cells in Patients with EAHEC O104:H4 Infection;PLoS ONE;2013-09-23
3. Inherited Complement Deficiencies;Emery and Rimoin's Principles and Practice of Medical Genetics;2013
4. Structural remodeling, trafficking and functions of glycosylphosphatidylinositol-anchored proteins;Progress in Lipid Research;2011-10
5. GPI-Anchor: Update for Biosynthesis and Remodeling;Trends in Glycoscience and Glycotechnology;2010
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