Proto-oncogene c-junand c-fosmessenger RNAs increase in the liver of carnitine-deficient juvenile visceral steatosis (jvs) mice
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(92)81368-V/fullpdf
Reference32 articles.
1. Infantile disease with microvesicular fatty infiltration of viscera spontaneously occurring in the C3H-H-2° strain of mouse with similarities to Reye's syndrome
2. Urea cycle disorder in C3H-H-2° mice with juvenile steatosis of viscera
3. Animal model of systemic carnitine deficiency: Analysis in C3H-H-2° strain of mouse associated with juvenile visceral steatosis
4. Carnitine administration to juvenile visceral steatosis mice corrects the suppressed expression of urea cycle enzymes by normalizing their transcription.
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1. Dietary L-carnitine alters gene expression in skeletal muscle of piglets;Molecular Nutrition & Food Research;2010-10-11
2. The carnitine system and body composition;Acta Diabetologica;2003-10
3. Hyperammonemia in carnitine-deficient adult JVS mice caused by starvation;Metabolic Brain Disease;2002
4. Antagonizing Effect of AP-1 on Glucocorticoid Induction of Urea Cycle Enzymes: A Study of Hyperammonemia in Carnitine-Deficient, Juvenile Visceral Steatosis Mice;Molecular Genetics and Metabolism;2000-12
5. Involvement of a cis-Acting Element in the Suppression of Carbamoyl Phosphate Synthetase I Gene Expression in the Liver of Carnitine-Deficient Mice;Molecular Genetics and Metabolism;1999-11
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