Electrophysiological properties of the hypokalaemic periodic paralysis mutation (R528H) of the skeletal muscle α 1S subunit as expressed in mouse L cells-Reference-Cited by-同舟云学术

Electrophysiological properties of the hypokalaemic periodic paralysis mutation (R528H) of the skeletal muscle α 1S subunit as expressed in mouse L cells

Published:1996-03-18 Issue:3 Volume:382 Page:244-248
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Lapie Pascale,Goudet Cyril,Nargeot Joël,Fontaine Bertrand,Lory Philippe

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(96)00173-1/fullpdf

Reference26 articles.

1. Mapping of the hypokalaemic periodic paralysis (HypoPP) locus to chromosome 1q31–32 in three European families

2. Structure and Function of Voltage-Sensitive Ion Channels

3. A calcium channel mutation causing hypokalemic periodic paralysis

4. Dihydropyridine receptor mutations cause hypokalemic periodic paralysis

5. Structure and function of voltage-gated ion channels

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2. Targeted Therapies for Skeletal Muscle Ion Channelopathies: Systematic Review and Steps Towards Precision Medicine;Journal of Neuromuscular Diseases;2021-05-13

3. Skeletal muscle CaV1.1 channelopathies;Pflügers Archiv - European Journal of Physiology;2020-03-28

4. When muscle Ca2+ channels carry monovalent cations through gating pores: insights into the pathophysiology of type 1 hypokalaemic periodic paralysis;The Journal of Physiology;2018-04-15

5. Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels;Journal of General Physiology;2018-01-31