Effects of hypothalamic peptides on electrical activity and membrane currents of ‘patch perforated’ clamped GH3anterior pituitary cells
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(91)80243-V/fullpdf
Reference26 articles.
1. Somatostatin Blocks Ca2+Action Potential Activity in Prolactin-Secreting Pituitary Tumor Cells through Coordinate Actions on K+ and Ca2+Conductances*
2. Membrane potential changes caused by thyrotropin-releasing hormone in the clonal GH3 cell and their relationship to secretion of pituitary hormone.
3. Biphasic effect of thyrotropin-releasing hormone on membrane K+ permeability in rat clonal pituitary cells
4. Rundown of GH3 cell K+ conductance response to TRH following patch recording can be obviated with GH3 cell extract
5. Electrophysiology of excitable endocrine cells
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1. Estradiol-modified prolactin secretion independently of action potentials and Ca2+ and blockade of outward potassium currents in GH3 cells;Naunyn-Schmiedeberg's Archives of Pharmacology;2016-10-17
2. Androgens block outward potassium currents and decrease spontaneous action potentials in GH3 cells;Naunyn-Schmiedeberg's Archives of Pharmacology;2014-10-26
3. Cell type influences the molecular mechanisms involved in hormonal regulation of ERG K+ channels;Pflügers Archiv - European Journal of Physiology;2012-03-14
4. An update on somatostatin receptor signaling in native systems and new insights on their pathophysiology;Pharmacology & Therapeutics;2007-11
5. Specificity of TRH receptor coupling to G-proteins for regulation of ERG K+channels in GH3rat anterior pituitary cells;The Journal of Physiology;2005-07-27
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