Characterization of potassium channels in pancreatic β cells from ob /ob mice
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(90)81518-S/fullpdf
Reference18 articles.
1. Cyclic changes in potential and resistance of the beta-cell membrane induced by glucose in islets of Langerhans from mouse.
2. Significance of ionic fluxes and changes in membrane potential for stimulus-secretion coupling in pancreatic B-cells
3. Defective ion transport in diabetic mouse islet cells
4. The electrical activity of pancreatic β-cells of diabetic mice
5. MEMBRANE POTENTIAL MEASUREMENTS IN ISLETS OF LANGERHANS FROMob/obOBESE MICE SUGGEST AN ALTERATION IN [Ca2+]i-ACTIVATED K+PERMEABILITY
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1. Impact of biotin supplemented diet on mouse pancreatic islet β-cell mass expansion and glucose induced electrical activity;Islets;2022-06-27
2. Pancreatic Islets from Hypothalamic Obese Rats Maintain K+ATP Channel-Dependent but Not -Independent Pathways on Glucose-Induced Insulin Release Process;Endocrine;2006
3. The adipoinsular axis: effects of leptin on pancreatic β-cells;American Journal of Physiology-Endocrinology and Metabolism;2000-01-01
4. Leptin suppression of insulin secretion by the activation of ATP-sensitive K+ channels in pancreatic beta-cells;Diabetes;1997-06-01
5. Differential modulation of pancreatic β-cell bursting by intracellular pH in the presence and absence of a K-ATP channel blocker;FEBS Letters;1993-10-11
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