STE2/SCG1-dependent inhibition of STE4-induced growth arrest by mutant STE4ΔC6 in the yeast pheromone response pathway
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(95)00526-F/fullpdf
Reference22 articles.
1. Phermone response in yeast
2. The protein kinase homologue Ste20p is required to link the yeast pheromone response G-protein beta gamma subunits to downstream signalling components.
3. Signal transduction in yeast mating: Receptors, transcription factors, and the kinase connection
4. FUS3 encodes a cdc2+/CDC28-related kinase required for the transition from mitosis into conjugation
5. Identification of a gene necessary for cell cycle arrest by a negative growth factor of yeast: FAR1 is an inhibitor of a G1 cyclin, CLN2
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1. The Gβ(KlSte4p) subunit of the heterotrimeric G protein has a positive and essential role in the induction of mating in the yeastKluyveromyces lactis;Yeast;2005
2. Modular Design of Gβ as the Basis for Reversible Specificity in Effector Stimulation;Journal of Biological Chemistry;2001-09
3. The Leu-132 of the Ste4(Gβ) subunit is essential for proper coupling of the G protein with the Ste2 α factor receptor during the mating pheromone response in yeast;FEBS Letters;2000-02-02
4. A C-terminal Mutant of the G Protein β Subunit Deficient in the Activation of Phospholipase C-β;Journal of Biological Chemistry;1996-08
5. Separate roles for N- and C-termini of the STE4 (β) subunit of the Saccharomyces cerevisiae G protein in the mediation of the growth arrest. Lack of growth-arresting activity of mammalian βγ complexes;Yeast;1996-01
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