Increase in CO3 H- influx and cellular pH in glucose-stimulated pancreatic islets
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(83)80882-5/fullpdf
Reference20 articles.
1. Insulin release: The fuel hypothesis
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3. The Stimulus-Secretion Coupling of Glucose-Induced Insulin Release - II. Interaction of Alkali and Alkaline Earth Cations
4. Stimulus-secretion coupling of glucose-induced insulin release. Effect of intracellular acidification upon calcium efflux from islet cells
5. The Stimulus-Secretion Coupling of Glucose-Induced Insulin Release. XLII. Effects of Extracellular pH on Insulin Release: Their Dependency on Nutrient Concentration
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2. Expression of TMEM16A and SLC4A4 in Human Pancreatic Islets;Cellular Physiology and Biochemistry;2012
3. Possible role of carbonic anhydrase in rat pancreatic islets: enzymatic, secretory, metabolic, ionic, and electrical aspects;American Journal of Physiology-Endocrinology and Metabolism;2007-06
4. Alkali pH directly activates ATP-sensitive K+ channels and inhibits insulin secretion in β-cells;Biochemical and Biophysical Research Communications;2006-11
5. Effects of Cholinergic M-Receptor Agonists on Insulin Release in Islets From Obese and Lean Mice of Different Ages;Pancreas;2004-11
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