Retinoic acid inhibits phospholipid turnover and protein kinase C activity in RA-sensitive but not in RA-resistant cells
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0014-5793(88)81050-0/fullpdf
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1. Ability of oncogenically transformed cells to grow without anchorage correlates with phosphorylation of a group of cell surface membrane proteins
2. Differential response to retinoic acid of Syrian hamster embryo fibroblasts expressing v-src or v-Ha-ras oncogenes.
3. Thrombin-induced phosphodiesteratic cleavage of phosphatidylinositol bisphosphate in human platelets.
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1. Alteration of Neuroblastoma Ganglioside Metabolism by Retinoic Acid;Journal of Neurochemistry;2006-10-05
2. Pharmacologic Hemoglobin Reversal: The Importance of Lipid Intermediaries and the Proposed Involvement of the Camp and Phosphatidylinositol Second Messenger Systems;Hemoglobin;1998-01
3. Retinoic acid induces translocation of protein kinase C (PKC) and activation of nuclear PKC (nPKC) in rat splenocytes;International Journal of Immunopharmacology;1995-04
4. Involvement of protein kinase C in the axonal growth-promoting effect on spinal cord neurons by target-derived astrocytes;Journal of Neurobiology;1994-12
5. Effects of retinoic acid on NB 69 human neuroblastoma cells and fetal rat mid brain neurons;Journal of Neural Transmission - Parkinsons Disease and Dementia Section;1994-02
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