Plasma biomarkers of liver injury and inflammation demonstrate a lack of apoptosis during obstructive cholestasis in mice-Reference-Cited by-同舟云学术

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Plasma biomarkers of liver injury and inflammation demonstrate a lack of apoptosis during obstructive cholestasis in mice

Published:2013-12 Issue:3 Volume:273 Page:524-531
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Woolbright Benjamin L.,Antoine Daniel J.,Jenkins Rosalind E.,Bajt Mary Lynn,Park B. Kevin,Jaeschke Hartmut

Funder

National Institutes of Health

National Center for Research Resources

National Institute of General Medical Sciences

Institutional Development Award (IDeA)

Training Program in Environmental Toxicology

National Institute of Environmental Health Sciences

Medical Research Council

Wellcome Trust Research Fellowship

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference58 articles.

1. Bile acids induce inflammatory genes in hepatocytes: a novel mechanism of inflammation during obstructive cholestasis;Allen;Am. J. Pathol.,2011

2. High-mobility group box-1 protein and keratin-18, circulating serum proteins informative of acetaminophen-induced necrosis and apoptosis in vivo;Antoine;Toxicol. Sci.,2009

3. Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity;Antoine;Mol. Med.,2010

4. Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity;Antoine;J. Hepatol.,2012

5. Mechanistic biomarkers provide early and sensitive detection of acetaminophen-induced acute liver injury at first presentation to hospital;Antoine;Hepatology,2013

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