The spleen as an extramedullary source of inflammatory cells responding to acetaminophen-induced liver injury
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Pharmacology,Toxicology
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1. In vivo upstream factors of mouse hepatotoxic mechanism with sustained hepatic glutathione depletion: Acetaminophen metabolite-erythrocyte adducts and splenic macrophage-generated reactive oxygen species;Chemico-Biological Interactions;2024-08
2. Alteration of Spleen Macrophage Phenotypes Impacts Exosomes-Related Proteins in Gentamicin Toxicity Model;J BIOL REG HOMEOS AG;2024
3. Intravital imaging of splenic classical monocytes modifying the hepatic CX3CR1+ cells motility to exacerbate liver fibrosis via spleen-liver axis;Theranostics;2024
4. Toxicant-induced injury and tissue repair;Essentials of Pharmatoxicology in Drug Research, Volume 1;2023
5. The versatility of macrophage heterogeneity in liver fibrosis;Frontiers in Immunology;2022-08-05
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