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Gambogic acid attenuates liver fibrosis by inhibiting the PI3K/AKT and MAPK signaling pathways via inhibiting HSP90

  • Published:2019-05 Issue: Volume:371 Page:63-73
  • ISSN:0041-008X
  • Container-title:Toxicology and Applied Pharmacology
  • language:en
  • Short-container-title:Toxicology and Applied Pharmacology

Author:

Yu Zhenlong,Jv Yanan,Cai Lu,Tian Xiangge,Huo Xiaokui,Wang Chao,Zhang Baojing,Sun ChengPeng,Ning Jing,Feng Lei,Zhang HouliORCID,Ma Xiaochi

Funder

National Natural Science Foundation of China

Liaoning Provincial Natural Science Foundation

Distinguished professor of Liaoning Province

Liaoning BaiQianWan Talents Program

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference28 articles.

1. The heat shock protein 90 inhibitor, 17-AAG, attenuates thioacetamide induced liver fibrosis in mice;Abu-Elsaad;Pharmacol. Rep.,2016

2. Inhibition of heat shock protein (molecular weight 90 kDa) attenuates proinflammatory cytokines and prevents lipopolysaccharide-induced liver injury in mice;Ambade;Hepatology,2012

3. Decoding cell death signals in liver inflammation;Brenner;J. Hepatol.,2013

4. Phytochemical constituents and pharmacological properties of Garcinia xanthochymus- a review;Che Hassan;Biomed. Pharmacother.,2018

5. An open-labeled, randomized, multicenter phase IIa study of gambogic acid injection for advanced malignant tumors;Chi;Chin. Med. J.,2013
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