Carbon tetrachloride induced mitochondrial division, respiratory chain damage, abnormal intracellular [H+] and apoptosis are due to the activation of 5-HT degradation system in hepatocytes-Reference-Cited by-同舟云学术

Carbon tetrachloride induced mitochondrial division, respiratory chain damage, abnormal intracellular [H+] and apoptosis are due to the activation of 5-HT degradation system in hepatocytes

Published:2022-03 Issue: Volume:439 Page:115929
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Zhang Yuxin,Liang Xiurui,Guan Jing,Jin Jiaqi,Zhang Yi,Xu Fan,Fu Jihua

Funder

National Natural Science Foundation of China National Outstanding Youth Science Fund Project

National Natural Science Foundation of China

China Pharmaceutical University

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

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2. Profile of 5-HT2A receptor involved in signaling cascades associated to intracellular inflammation and apoptosis in hepatocytes and its role in carbon tetrachloride-induced hepatotoxicity;Cellular Signalling;2023-05

3. Myocardial ischemia-reperfusion injury is probably due to the excessive production of mitochondrial ROS caused by the activation of 5-HT degradation system mediated by PAF receptor;Molecular Immunology;2023-03

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5. Corrigendum to “Carbon tetrachloride induced mitochondrial division, respiratory chain damage, abnormal intracellular [H+] and apoptosis are due to the activation of 5-HT degradation system in hepatocytes” [Toxicology and Applied Pharmacology, 439C (2022), 115929];Toxicology and Applied Pharmacology;2022-06