Prenatal administration of the cytochrome P4501A inducer, Β-naphthoflavone (BNF), attenuates hyperoxic lung injury in newborn mice: Implications for bronchopulmonary dysplasia (BPD) in premature infants

Author:

Couroucli Xanthi I.,Liang Yan-hong Wei,Jiang Weiwu,Wang Lihua,Barrios Roberto,Yang Peiying,Moorthy Bhagavatula

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference69 articles.

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3. Long-term (imprinting) effects of transplacental treatment of mice with 3-methylcholanthrene or beta-naphthoflavone on hepatic metabolism of 3-methylcholanthrene;Anderson;Pharmacol. Toxicol.,1991

4. Repression of cytochrome P450 1A1 gene expression by oxidative stress: mechanisms and biological implications;Barouki;Biochem. Pharmacol.,2001

5. Regulation of cytochrome P4501A1 expression by hyperoxia in human lung cell lines: Implications for hyperoxic lung injury;Bhakta;Toxicol. Appl. Pharmacol.,2008

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