In utero exposure to dipentyl phthalate disrupts fetal and adult Leydig cell development-Reference-Cited by-同舟云学术

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In utero exposure to dipentyl phthalate disrupts fetal and adult Leydig cell development

Published:2021-05 Issue: Volume:419 Page:115514
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Chen Haiqiong,Xin Xiu,Liu Miaoqing,Ma Feifei,Yu Yige,Huang Jie,Dai Haipeng,Li Zhongrong,Ge Ren-shan

Funder

National Natural Science Foundation of China

Health Commission of Zhejiang Province

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference60 articles.

1. A cell-autonomous molecular cascade initiated by AMP-activated protein kinase represses steroidogenesis;Abdou;Mol. Cell. Biol.,2014

2. Steroidogenic acute regulatory protein (StAR) retains activity in the absence of its mitochondrial import sequence: implications for the mechanism of StAR action;Arakane;Proc. Natl. Acad. Sci. U. S. A.,1996

3. Sperm function in Indonesian men treated with testosterone enanthate;Arsyad;Int. J. Androl.,1993

4. Influence of in utero di-n-hexyl phthalate and dicyclohexyl phthalate on fetal testicular development in rats;Aydogan Ahbab;Toxicol. Lett.,2015

5. Simvastatin and dipentyl phthalate lower ex vivo testicular testosterone production and exhibit additive effects on testicular testosterone and gene expression via distinct mechanistic pathways in the fetal rat;Beverly;Toxicol. Sci.,2014

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