Endothelial (NOS3 E298D) and inducible (NOS2 exon 22) nitric oxide synthase polymorphisms, as well as plasma NOx, influence sepsis development

Author:

Martin Guadalupe,Asensi Víctor,Montes A. Hugo,Collazos Julio,Alvarez Victoria,Pérez-Is Laura,Carton José A.,Taboada Francisco,Valle-Garay Eulalia

Funder

Oviedo University

Janssen Pharmaceuticals and Red Temática de Investigación de SIDA (RIS), Instituto de Salud Carlos III (ISCIII), Plan Estatal I + D + i & European Regional Development Fund (ERDF) “Una manera de hacer Europa”

Publisher

Elsevier BV

Subject

Cancer Research,Clinical Biochemistry,Physiology,Biochemistry

Cited by 10 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Kinetics of Biomarkers of Oxidative Stress in Septic Shock: A Pilot Study;Antioxidants;2022-03-26

2. HDL and Sepsis;HDL Metabolism and Diseases;2022

3. Renal microvascular endothelial cell responses in sepsis-induced acute kidney injury;Nature Reviews Nephrology;2021-10-19

4. Variants in TNF and NOS3 ( eNOS ) genes associated with sepsis in adult patients;The Journal of Gene Medicine;2021-02-26

5. Endothelial Responses in Sepsis;American Journal of Respiratory and Critical Care Medicine;2020-08-01

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