Urolithin A attenuates osteoclast differentiation and compensates for ovariectomy-induced bone loss in mice by inhibiting PI3K/AKT/mTOR signaling pathway-Reference-Cited by-同舟云学术

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Urolithin A attenuates osteoclast differentiation and compensates for ovariectomy-induced bone loss in mice by inhibiting PI3K/AKT/mTOR signaling pathway

Published:2024-02 Issue:1 Volume:4 Page:100495
ISSN:2667-0313
Container-title:Phytomedicine Plus
language:en
Short-container-title:Phytomedicine Plus

Author:

Zhou Wenyun,Zhou Weijun,Zhou Zibin,Bi Yonghao,Zhou Zhigao,Chen Shaozhe,Lin Zihong,Ye Jiajie,Xie Gang,Liu Wenjie,Xie Dantao,Lian Zhen,Zheng Ruiwu,Hu Xianghua,Wang Xinjia,Yuan Guixin,Yao Guanfeng

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Complementary and alternative medicine,General Pharmacology, Toxicology and Pharmaceutics

Reference78 articles.

1. Osteocyte necrosis triggers osteoclast-mediated bone loss through macrophage-inducible C-type lectin;Andreev;J. Clin. Investig.,2020

2. Allicin attenuates pathological cardiac hypertrophy by inhibiting autophagy via activation of PI3K/Akt/mTOR and MAPK/ERK/mTOR signaling pathways;Ba;Phytomedicine,2019

3. Osteoclast differentiation and activation;Boyle;Nature,2003

4. Protein kinase B (c-Akt) in phosphatidylinositol-3-OH kinase signal transduction;Burgering;Nature,1995

5. The metabolite urolithin-A ameliorates oxidative stress in neuro-2a cells, becoming a potential neuroprotective agent;Casedas;Antioxidants,2020

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