Iron overload in hereditary tyrosinemia type 1 induces liver injury through the Sp1/Tfr2/hepcidin axis-Reference-Cited by-同舟云学术

Iron overload in hereditary tyrosinemia type 1 induces liver injury through the Sp1/Tfr2/hepcidin axis

Published:2016-07 Issue:1 Volume:65 Page:137-145
ISSN:0168-8278
Container-title:Journal of Hepatology
language:en
Short-container-title:Journal of Hepatology

Author:

Bao Wen-Dai,Fan Yao,Deng Yue-Zhen,Long Ling-Yun,Wang Jing-jing,Guan Dong-Xian,Qian Zhen-Yu,An Peng,Feng Yuan-Yuan,He Zhi-Ying,Wang Xiao-Fan,Phillip Koeffler H.,Hu Ronggui,Wang Jianshe,Wang Xin,Wang Fudi,Li Jing-Jing,Xie Dong

Publisher

Elsevier BV

Subject

Hepatology

Reference37 articles.

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3. Mutations in HFE2 cause iron overload in chromosome 1q-linked juvenile hemochromatosis;Papanikolaou;Nat Genet,2004

4. Mutant antimicrobial peptide hepcidin is associated with severe juvenile hemochromatosis;Roetto;Nat Genet,2003

5. The gene TFR2 is mutated in a new type of haemochromatosis mapping to 7q22;Camaschella;Nat Genet,2000

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