Modeling the complex pathology of Alzheimer's disease in Drosophila-Reference-Cited by-同舟云学术

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Modeling the complex pathology of Alzheimer's disease in Drosophila

Published:2015-12 Issue: Volume:274 Page:58-71
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Fernandez-Funez Pedro,de Mena Lorena,Rincon-Limas Diego E.

Funder

Florida Department of Health

UF department of Neurology

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference163 articles.

1. Retro-inversal of intracellular selected beta-amyloid-interacting peptides: implications for a novel Alzheimer's disease treatment;Acerra;Biochemistry,2014

2. The genome sequence of Drosophila melanogaster;Adams;Science,2000

3. NMNAT suppresses tau-induced neurodegeneration by promoting clearance of hyperphosphorylated tau oligomers in a Drosophila model of tauopathy;Ali;Hum. Mol. Genet.,2012

4. A commonly used Drosophila model of Alzheimer's disease generates an aberrant species of amyloid-beta with an additional N-terminal glutamine residue;Allan;FEBS Lett.,2014

5. Functional genomic screen and network analysis reveal novel modifiers of tauopathy dissociated from tau phosphorylation;Ambegaokar;Hum. Mol. Genet.,2011

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