Demyelination as a rational therapeutic target for ischemic or traumatic brain injury-Reference-Cited by-同舟云学术

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Demyelination as a rational therapeutic target for ischemic or traumatic brain injury

Published:2015-10 Issue: Volume:272 Page:17-25
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Shi Hong,Hu Xiaoming,Leak Rehana K.,Shi Yejie,An Chengrui,Suenaga Jun,Chen Jun,Gao Yanqin

Funder

Natural Science Foundation of Shanghai

Natural Science Foundation of China

Shanghai Committee of Science and Technology Research Program

American Heart Association Scientist Development

Chinese Ministry of Education

NIH/NINDS

Department of Veterans Affairs Research Career Scientist Award

RR & D Merit Review

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference178 articles.

1. Ca(2+) influx through AMPA or kainate receptors alone is sufficient to initiate excitotoxicity in cultured oligodendrocytes;Alberdi;Neurobiol. Dis.,2002

2. Genetic background determines phenotypic severity of the Plp rumpshaker mutation;Al-Saktawi;J. Neurosci. Res.,2003

3. Metabolic aspects of neuron–oligodendrocyte–astrocyte interactions;Amaral;Front. Endocrinol. (Lausanne),2013

4. Molecular dialogs between the ischemic brain and the peripheral immune system: dualistic roles in injury and repair;An;Prog. Neurobiol.,2014

5. Integrin dynamics produce a delayed stage of long-term potentiation and memory consolidation;Babayan;J. Neurosci.,2012

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