Neurogranin restores amyloid β-mediated synaptic transmission and long-term potentiation deficits-Reference-Cited by-同舟云学术

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Neurogranin restores amyloid β-mediated synaptic transmission and long-term potentiation deficits

Published:2016-03 Issue: Volume:277 Page:115-123
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Kaleka Kanwardeep Singh,Gerges Nashaat Z.

Funder

National Institute on Aging

BrightFocus Foundation

Advancing a Healthier Wisconsin

Alzheimer's Association

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference65 articles.

1. Wnt-5a occludes Abeta oligomer-induced depression of glutamatergic transmission in hippocampal neurons;Cerpa;Mol. Neurodegener.,2010

2. Dendritic translocation of RC3/neurogranin mRNA in normal aging, Alzheimer disease and fronto-temporal dementia;Chang;J. Neuropathol. Exp. Neurol.,1997

3. Action of amyloid beta-protein on protein kinase C activity;Chauhan;Life Sci.,1991

4. Neurochemical dissection of synaptic pathology in Alzheimer's disease;Davidsson;Int. Psychogeriatr.,1998

5. N-methyl-d-aspartate receptors are required for synaptic targeting of Alzheimer's toxic amyloid-β peptide oligomers;Decker;J. Neurochem.,2010

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