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Neuronal-specific impairment of heparan sulfate degradation in Drosophila reveals pathogenic mechanisms for Mucopolysaccharidosis type IIIA

  • Published:2018-05 Issue: Volume:303 Page:38-47
  • ISSN:0014-4886
  • Container-title:Experimental Neurology
  • language:en
  • Short-container-title:Experimental Neurology

Author:

Webber Dani L.,Choo Amanda,Hewson Laura J.,Trim Paul J.,Snel Marten F.,Hopwood John J.,Richards Robert I.,Hemsley Kim M.,O'Keefe Louise V.

Funder

University of Adelaide

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference80 articles.

1. Sanfilippo syndrome: overall review;Andrade;Pediatr. Int.,2015

2. Neuroinflammatory and oxidative stress phenomena in MPS IIIA mouse model: the positive effect of long-term aspirin treatment;Arfi;Mol. Genet. Metab.,2011

3. An improved method for glycosaminoglycan analysis by LC-MS/MS of urine samples collected on filter paper;Auray-Blais;Clin. Chim. Acta,2012

4. Functional binding of secreted molecules to heparan sulfate proteoglycans in Drosophila;Baeg;Curr. Opin. Cell Biol.,2000

5. Heparan sulfate proteoglycans are critical for the organization of the extracellular distribution of wingless;Baeg;Development,2001

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