Commentary on Giralt et al.: PTK2B/Pyk2 overexpression improves a mouse model of Alzheimer's disease
Author:
Funder
Israel Cancer Association
Estee Lauder Companies
Israel Science Foundation
Israel Cancer Research Fund
Publisher
Elsevier BV
Subject
Developmental Neuroscience,Neurology
Reference29 articles.
1. Alzheimer's Disease Facts and Figures;Alzheimer's Association,2017
2. Postsynaptic clustering and activation of Pyk2 by PSD-95;Bartos;J Neurosci,2010
3. CD33 modulates TREM2: convergence of Alzheimer loci;Chan;Nat Neurosci,2015
4. Functional screening of Alzheimer risk loci identifies PTK2B as an in vivo modulator and early marker of Tau pathology;Dourlen;Mol Psychiatry,2017
5. Pyk2 modulates hippocampal excitatory synapses and contributes to cognitive deficits in a Huntington's disease model;Giralt;Nat Commun,2017
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1. FAK-Mediated Signaling Controls Amyloid Beta Overload, Learning and Memory Deficits in a Mouse Model of Alzheimer’s Disease;International Journal of Molecular Sciences;2022-08-13
2. Pyk2 Stabilizes Striatal Medium Spiny Neuron Structure and Striatal-Dependent Action;Cells;2021-12-07
3. Inhibition of soluble epoxide hydrolase (sEH) protects hippocampal neurons and reduces cognitive decline in type 2 diabetic mice;European Journal of Neuroscience;2021-03-22
4. 14,15-Epoxyeicosatrienoic Acid Alleviates Pathology in a Mouse Model of Alzheimer's Disease;The Journal of Neuroscience;2020-09-24
5. Tau Interacting Proteins: Gaining Insight into the Roles of Tau in Health and Disease;Advances in Experimental Medicine and Biology;2019
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