A commentary on: Overexpression of Dyrk1A inhibits choline acetyltransferase induction by oleic acid in cellular models of Down syndrome
Author:
Funder
Jérôme Lejeune Foundation
Catalan Agency for Administration of University and Research
Spanish Ministry of Education and Sciences
EU (CureFXS)
Publisher
Elsevier BV
Subject
Developmental Neuroscience,Neurology
Reference44 articles.
1. Neurodevelopmental delay, motor abnormalities and cognitive deficits in transgenic mice overexpressing Dyrk1A (minibrain), a murine model of Down's syndrome;Altafaj;Hum. Mol. Genet.,2001
2. Increased NR2A expression and prolonged decay of NMDA-induced calcium transient in cerebellum of TgDyrk1A mice, a mouse model of Down syndrome;Altafaj;Neurobiol. Dis.,2008
3. Oleic acid ameliorates amyloidosis in cellular and mouse models of Alzheimer's disease;Amtul;Brain Pathol,2011
4. Impaired spatial learning strategies and novel object recognition in mice haploinsufficient for the dual specificity tyrosine-regulated kinase-1A (Dyrk1A);Arque;PLoS One,2008
5. Dyrk1A is dynamically expressed on subsets of motor neurons and in the neuromuscular junction: possible role in Down syndrome;Arque;PLoS One,2013
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