Impairment of pericyte-endothelium crosstalk leads to blood-brain barrier dysfunction following traumatic brain injury-Reference-Cited by-同舟云学术

Impairment of pericyte-endothelium crosstalk leads to blood-brain barrier dysfunction following traumatic brain injury

Published:2019-07 Issue: Volume:317 Page:260-270
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Bhowmick Saurav,D'Mello Veera,Caruso Danielle,Wallerstein Alex,Abdul-Muneer P.M.^ORCID

Funder

Neuroscience Institute at Hackensack Meridian Health JFK Medical Center

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference59 articles.

1. The mechanisms of cerebral vascular dysfunction and neuroinflammation by MMP-mediated degradation of VEGFR-2 in alcohol ingestion;Abdul Muneer;Arterioscler. Thromb. Vasc. Biol.,2012

2. Induction of oxidative and nitrosative damage leads to cerebrovascular inflammation in an animal model of mild traumatic brain injury induced by primary blast;Abdul-Muneer;Free Radic. Biol. Med.,2013

3. Activation of NLRP3 inflammasome by cholesterol crystals in alcohol consumption induces atherosclerotic lesions;Abdul-Muneer;Brain Behav. Immun.,2017

4. Angiotensin II causes neuronal damage in stretch-injured neurons: protective effects of losartan, an angiotensin T1 receptor blocker;Abdul-Muneer;Mol. Neurobiol.,2017

5. Traumatic brain injury induced matrix metalloproteinase2 cleaves CXCL12alpha (stromal cell derived factor 1alpha) and causes neurodegeneration;Abdul-Muneer;Brain Behav. Immun.,2017

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