EPPS treatment attenuates traumatic brain injury in mice by reducing Aβ burden and ameliorating neuronal autophagic flux-Reference-Cited by-同舟云学术

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EPPS treatment attenuates traumatic brain injury in mice by reducing Aβ burden and ameliorating neuronal autophagic flux

Published:2019-04 Issue: Volume:314 Page:20-33
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Anthony Jalin Angela Melinda A.,Jin Rong,Wang Min,Li Guohong

Funder

National Institutes of Health

Louisiana State University

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference50 articles.

1. Synaptic activity protects against AD and FTD-like pathology via autophagic-lysosomal degradation;Akwa;Mol. Psychiatry,2018

2. Traumatic brain injury pathophysiology and treatments: early, intermediate, and late phases post-injury;Algattas;Int. J. Mol. Sci.,2013

3. Autophagy biomarkers Beclin 1 and p62 are increased in cerebrospinal fluid after traumatic brain injury;Au;Neurocrit. Care.,2017

4. Autophagy in neurodegenerative disorders: pathogenic roles and therapeutic implications;Banerjee;Trends Neurosci.,2010

5. A new device for experimental modeling of central nervous system injuries;Bilgen;Neurorehabil. Neural Repair,2005

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3. Impaired autophagic flux in the human brain after traumatic brain injury;NeuroReport;2024-03-01

4. HSPB2 facilitates neural regeneration through autophagy for sensorimotor recovery after traumatic brain injury;JCI Insight;2023-08-22

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