Facilitation of distinct inhibitory synaptic inputs by chemical anoxia in neurons in the oculomotor, facial and hypoglossal motor nuclei of the rat-Reference-Cited by-同舟云学术

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Facilitation of distinct inhibitory synaptic inputs by chemical anoxia in neurons in the oculomotor, facial and hypoglossal motor nuclei of the rat

Published:2017-04 Issue: Volume:290 Page:95-105
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Takagi Satoshi,Kono Yu,Nagase Masashi,Mochio Soichiro,Kato Fusao

Funder

Ministry of Education, Culture, Sports, Science and Technology, Japan

Japan Health Sciences Foundation

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference42 articles.

1. The effect of simulated ischaemia on spontaneous GABA release in area CA1 of the juvenile rat hippocampus;Allen;J. Physiol.,2004

2. The transporters GlyT2 and VIAAT cooperate to determine the vesicular glycinergic phenotype;Aubrey;J. Neurosci.,2007

3. Development of GABAergic and glycinergic transmission in the neonatal rat dorsal horn;Baccei;J. Neurosci.,2004

4. Protective role of neuronal KATP channels in brain hypoxia;Ballanyi;J. Exp. Biol.,2004

5. Neurochemical evidence that glycine induces bioenergetical dysfunction;Busanello;Neurochem. Int.,2010

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1. Developmental nicotine exposure alters glycinergic neurotransmission to hypoglossal motoneurons in neonatal rats;Journal of Neurophysiology;2018-09-01

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