Targeting the JAK2-STAT3 pathway to inhibit cGAS-STING activation improves neuronal senescence after ischemic stroke-Reference-Cited by-同舟云学术

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Targeting the JAK2-STAT3 pathway to inhibit cGAS-STING activation improves neuronal senescence after ischemic stroke

Published:2023-10 Issue: Volume:368 Page:114474
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Zhang Wei,Xu Meijie,Chen Fangyu,Su Yue,Yu Mingjing,Xing Ling,Chang Yifan,Yan Tao

Funder

Tianjin Municipal Health Commission

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference54 articles.

1. cGAS in action: expanding roles in immunity and inflammation;Ablasser;Science,2019

2. Cellular senescence as a mechanism and target in chronic lung diseases;Barnes;Am. J. Respir. Crit. Care Med.,2019

3. NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injury;Barrett;J. Neuroinflammation,2017

4. Elimination of senescent cells by beta-galactosidase-targeted prodrug attenuates inflammation and restores physical function in aged mice;Cai;Cell Res.,2020

5. Inhibition of JAK-STAT signaling pathway alleviates age-related phenotypes in tendon stem/progenitor cells;Chen;Front. Cell Dev. Biol.,2021

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