Activation of spinal chemokine receptor CXCR3 mediates bone cancer pain through an Akt-ERK crosstalk pathway in rats-Reference-Cited by-同舟云学术

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Activation of spinal chemokine receptor CXCR3 mediates bone cancer pain through an Akt-ERK crosstalk pathway in rats

Published:2015-01 Issue: Volume:263 Page:39-49
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Guan Xue-Hai,Fu Qiao-Chu,Shi Dai,Bu Hui-Lian,Song Zhen-Peng,Xiong Bing-Rui,Shu Bin,Xiang Hong-Bing,Xu Bing,Manyande Anne,Cao Fei,Tian Yu-Ke

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference103 articles.

1. Molecular psychology: roles for the ERK MAP kinase cascade in memory;Adams;Annu. Rev. Pharmacol. Toxicol.,2002

2. CXCR3 surface expression in human airway epithelial cells: cell cycle dependence and effect on cell proliferation;Aksoy;Am. J. Physiol. Lung Cell. Mol. Physiol.,2006

3. Tissue plasminogen activator contributes to morphine tolerance and induces mechanical allodynia via astrocytic IL-1beta and ERK signaling in the spinal cord of mice;Berta;Neuroscience.,2013

4. Delayed functional expression of neuronal chemokine receptors following focal nerve demyelination in the rat: a mechanism for the development of chronic sensitization of peripheral nociceptors;Bhangoo;Mol. Pain,2007

5. Expression of CXCR3 on specific T cells is essential for homing to the prostate gland in an experimental model of chronic prostatitis/chronic pelvic pain syndrome;Breser;J. Immunol.,2013

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