Regional MeCP2 expression levels in the female MeCP2-deficient mouse brain correlate with specific behavioral impairments-Reference-Cited by-同舟云学术

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Regional MeCP2 expression levels in the female MeCP2-deficient mouse brain correlate with specific behavioral impairments

Published:2013-01 Issue: Volume:239 Page:49-59
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Wither Robert G.,Lang Min,Zhang Liang,Eubanks James H.

Funder

Canadian Institutes of Health Research

Ontario Rett Syndrome Association

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference53 articles.

1. The X-linked methylated DNA binding protein, MeCP2, is subject to X inactivation in the mouse;Adler;Mamm. Genome,1995

2. miRNA-132 orchestrates chromatin remodeling and translational control of the circadian clock;Alvarez-Saavedra;Hum. Mol. Genet.,2011

3. Rett syndrome is caused by mutations in X-linked MeCP2, encoding methyl-CpG-binding protein 2;Amir;Nat. Genet.,1999

4. Correlation between clinical severity in patients with Rett syndrome with a p.R168X or p.T158M MECP2 mutation, and the direction and degree of skewing of X-chromosome inactivation;Archer;J. Med. Genet.,2007

5. Hippocampal synaptic plasticity is impaired in the MeCP2-null mouse model of Rett Syndrome;Asaka;Neurobiol. Dis.,2006

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