Decreased expression of complement 3a receptor (C3aR) in human placentas from severe preeclamptic pregnancies
Author:
Publisher
Elsevier BV
Subject
Obstetrics and Gynecology,Reproductive Medicine
Reference43 articles.
1. Is inflammation the cause of pre-eclampsia?;Ramma;Biochemical Society Transactions,2011
2. Decidual hemostasis, inflammation, and angiogenesis in pre-eclampsia;Lockwood;Seminars in Thrombosis and Hemostasis,2011
3. C5a enhances dysregulated inflammatory and angiogenic responses to malaria in vitro: potential implications for placental malaria;Conroy;PLoS One,2009
4. Complement-mediated inhibition of neovascularization reveals a point of convergence between innate immunity and angiogenesis;Langer;Blood,2010
5. Regulation by complement C3a and C5a anaphylatoxins of cytokine production in human umbilical vein endothelial cells;Monsinjon;The FASEB Journal,2003
Cited by 12 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Utilizing Contrast-Enhanced Ultrasonography with Phosphatidylserine Microbubbles to Detect Placental Inflammation in Rhesus Macaques;Molecules;2023-03-23
2. Alternative pathway activation in pregnancy, a measured amount “complements” a successful pregnancy, too much results in adverse events;Immunological Reviews;2022-11-15
3. Identification of underlying mechanisms and hub gene-miRNA networks of the genomic subgroups in preeclampsia development;Medicine;2022-07-22
4. Mechanisms of Key Innate Immune Cells in Early- and Late-Onset Preeclampsia;Frontiers in Immunology;2020-08-18
5. Dysregulation of complement system during pregnancy in patients with preeclampsia: A prospective study;Molecular Immunology;2020-06
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3