Interferon-β, an autocrine cytokine, suppresses human fetal skin fibroblast migration into a denuded area in a cell monolayer but is not involved in the age-related decline of cell migration-Reference-Cited by-同舟云学术

Interferon-β, an autocrine cytokine, suppresses human fetal skin fibroblast migration into a denuded area in a cell monolayer but is not involved in the age-related decline of cell migration

Published:1996-06 Issue:3 Volume:87 Page:141-153
ISSN:0047-6374
Container-title:Mechanisms of Ageing and Development
language:en
Short-container-title:Mechanisms of Ageing and Development

Author:

Kondo Hiroshi,Yonezawa Yumiko,Ito Hideki

Publisher

Elsevier BV

Subject

Developmental Biology,Ageing

Reference33 articles.

1. Wound healing and aging;Goodson;J. Invest. Dermatol.,1979

2. Age-related differences in healing superficial skin wounds in humans;Grove;Arch. Dermatol. Res.,1982

3. Cell replacements in the human stratum corneum in old age;Baker;Rheumatology,1986

4. In vitro characterization of response to stimulus (wounding) with regard to ageing in human skin fibroblasts;Muggleton-Harris;Mech. Ageing Dev.,1982

5. Decline of fibroblast chemotaxis with age of donor and cell passage number;Albini;Collagen Rel. Res.,1988

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Interferon-β gene transfer inhibits melanoma cells adhesion and migration;Cytokine;2017-01

2. Interferon-β Inhibits Bleomycin-Induced Lung Fibrosis by Decreasing Transforming Growth Factor-β and Thrombospondin;American Journal of Respiratory Cell and Molecular Biology;2005-02

3. Impaired migration, integrin function, and actin cytoskeletal organization in dermal fibroblasts from a subset of aged human donors;Mechanisms of Ageing and Development;2001-08

4. Idiopathic Pulmonary Fibrosis: Prevailing and Evolving Hypotheses about Its Pathogenesis and Implications for Therapy;Annals of Internal Medicine;2001-01-16

5. Aging Fibroblasts Present Reduced Epidermal Growth Factor (EGF) Responsiveness Due to Preferential Loss of EGF Receptors;Journal of Biological Chemistry;2000-06