Repression of Transcriptional Activity of Estrogen Receptor a by α Cullin3/SPOP Ubiquitin E3 Ligase Complex
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,General Medicine
Reference20 articles.
1. Thyroid hormone is an inhibitor of estrogen-induced degradation of estrogen receptor-alpha protein: estrogen-dependent proteolysis is not essential for receptor transactivation function in the pituitary;Alarid;Endocrinology,2003
2. Inhibiting proteasomal proteolysis sustains estrogen receptor-alpha activation;Fan;Mol. Endocrinol,2004
3. Stable chromosome inactivation involves the PRC1 Polycomb complex and requires histone MACROH2A1 and the CULLIN3/SPOP ubiquitin E3 ligase;Hernandez-Munoz;Proc. Natl. Acad. Sci. USA,2005
4. Estrogen receptors and their downstream targets in cancer;Ikeda;Arch. Histol. Cytol,2004
5. Mitogenic estrogen metabolites alter the expression of 17beta-estradiol-regulated proteins including heat shock proteins in human MCF-7 breast cancer cells;Kim;Mol. Cells,2005
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