Neuronal degeneration and lipopigment formation in rat sympathetic ganglion after treatment with high-dose guanethidine
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference19 articles.
1. Microspectrofluorimetric quantitation of catecholamine fluorescence in rat sympathetic ganglia;Alho;Histochem. J.,1983
2. A new method of destroying adrenergic nerves in adult animals using guanethidine;Burnstock;Br. J. Pharmacol.,1971
3. Regional energy metabolism and lipofuscin accumulation in mouse brain during aging;Ferrendelli;J. Neuropathol. Exp. Neurol.,1971
4. Axon retraction following guanethidine treatment: studies of sympathetic neurons in tissue culture;Heath;J. Neurocytol.,1974
5. Selectivity of neuronal degeneration produced by chronic guanethidine treatment;Heath;J. Neurocytol.,1977
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3. The Lysosomal System in Neuronal Cell Death: A Review;Annals of the New York Academy of Sciences;1993-05
4. Effects of neonatal sympathectomy with 6-hydroxydopamine or guanethidine on survival of neurons in the intermediolateral cell column of rat spinal cord;Journal of the Autonomic Nervous System;1990-11
5. Suramin-induced changes in sympathetic neurons: Correlation between catecholamine fluorescence, tyrosine hydroxylase immunoreactivity and accumulation of pigment bodies;Neuroscience Letters;1990-04
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