Hypothalamic blood flow remains unaltered following chronic nitric oxide synthase blockade in rats
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference21 articles.
1. Nitric oxide (NO) is involved in increased cerebral blood flow following stimulation of the nucleus basalis of Meynert in anesthetized rats;Adachi;Neurosci. Lett.,1992
2. Determinants of aortic cyclic guanosine monophosphate in hypertension induced by chronic inhibition of nitric oxide synthase;Arnal;J. Clin. Invest.,1992
3. Measurement of local blood flow with hydrogen gas;Aukland;Circ. Res.,1964
4. Long term administration of l-arginine, l-NAME, and the exogenous NO donor molsidomine modulates urinary nitrate and cGMP excretion in rats;Böger;Cardiovasc. Res.,1994
5. Mono-l-arginine-containing compounds dilate piglet pial arterioles via an endothelium-derived relaxing factor-like substance;Busija;Circ. Res.,1990
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2. Hypersensitivity to Thromboxane Receptor Mediated Cerebral Vasomotion and CBF Oscillations during Acute NO-Deficiency in Rats;PLoS ONE;2010-12-31
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