Neural mechanisms mediating 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonism in the monkey: Relative contributions of the striatopallidal and striatonigral pathways as suggested by 2-deoxyglucose uptake

Author:

Mitchell I.J.,Cross A.J.,Sambrook M.A.,Crossman A.R.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference10 articles.

1. A primate model of parkinsonism: selective destruction of dopaminergic neurons in the pars compacta of the substantia nigra by N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine;Burns,1983

2. Identification of different populations of neostriatal neurones projecting to globus pallidus or substantia nigra in the monkey: a retrograde fluorescence double-labelling study;Feger;Neurosci. Lett.,1984

3. Parkinsonism: onset, progression and mortality;Hoehn;Neurology,1967

4. Local cerebral glucose utilization in the normal conscious macaque monkey;Kennedy;Ann. Neurol.,1978

5. Activity-dependent energy metabolism in rat posterior pituitary primarily reflects sodium pump activity;Mata;J. Neurochem.,1980

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