IL-10 disrupts the Brd4-docking sites to inhibit LPS-induced CXCL8 and TNF-α expression in monocytes: Implications for chronic obstructive pulmonary disease-Reference-Cited by-同舟云学术

IL-10 disrupts the Brd4-docking sites to inhibit LPS-induced CXCL8 and TNF-α expression in monocytes: Implications for chronic obstructive pulmonary disease

Published:2015-09 Issue:3 Volume:136 Page:781-791.e9
ISSN:0091-6749
Container-title:Journal of Allergy and Clinical Immunology
language:en
Short-container-title:Journal of Allergy and Clinical Immunology

Author:

Castellucci Monica,Rossato Marzia,Calzetti Federica,Tamassia Nicola,Zeminian Stefano,Cassatella Marco A.,Bazzoni Flavia^ORCID

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

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5. The primary mechanism of the IL-10-regulated antiinflammatory response is to selectively inhibit transcription;Murray;Proc Natl Acad Sci U S A,2005

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