Adhesion-induced eosinophil cytolysis requires the receptor-interacting protein kinase 3 (RIPK3)–mixed lineage kinase-like (MLKL) signaling pathway, which is counterregulated by autophagy-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Adhesion-induced eosinophil cytolysis requires the receptor-interacting protein kinase 3 (RIPK3)–mixed lineage kinase-like (MLKL) signaling pathway, which is counterregulated by autophagy

Published:2017-12 Issue:6 Volume:140 Page:1632-1642
ISSN:0091-6749
Container-title:Journal of Allergy and Clinical Immunology
language:en
Short-container-title:Journal of Allergy and Clinical Immunology

Author:

Radonjic-Hoesli Susanne^ORCID,Wang Xiaoliang,de Graauw Elisabeth,Stoeckle Christina,Styp-Rekowska Beata,Hlushchuk Ruslan,Simon Dagmar,Spaeth Peter J.,Yousefi Shida,Simon Hans-Uwe^ORCID

Funder

Swiss National Science Foundation

Foundation Allergiestiftung Ulrich Müller-Gierok, Bern

Swiss Cancer League

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference50 articles.

1. Eosinophilic disorders;Simon;J Allergy Clin Immunol,2007

2. Interactions between human eosinophils and schistosomula of Schistosoma mansoni. II. The mechanism of irreversible eosinophil adherence;Butterworth;J Exp Med,1979

3. Eosinophils as effector cells in immunity and hypersensitivity disorders;Kay;Clin Exp Immunol,1985

4. Ultrastructural characterization and morphometric analysis of eosinophil degranulation;Henderson;J Cell Sci,1985

5. Allergen-induced eosinophil cytolysis is a primary mechanism for granule protein release in human upper airways;Erjefält;Am J Respir Care Med,1999

Cited by 55 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Targeting necroptosis: a promising avenue for respiratory disease treatment;Cell Communication and Signaling;2024-08-28

2. The role of extracellular traps released by neutrophils, eosinophils, and macrophages in asthma;Respiratory Research;2024-07-30

3. Eosinophil granulocytes in chronic inflammatory respiratory diseases and CRSwNP: Function, immunological basis, and clinical significance;Allergologie select;2024-01-01

4. Eosinophils—from cradle to grave;Allergy;2023-09-13

5. Programmed Cell Death in Asthma: Apoptosis, Autophagy, Pyroptosis, Ferroptosis, and Necroptosis;Journal of Inflammation Research;2023-07

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3